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J Obes Eat Disord, 2017
ISSN: 2471-8203
August 23-24, 2017 | Toronto, Canada
allied
academies
INTERNATIONAL OBESITY, BARIATRIC AND
METABOLIC SURGERY SUMMIT AND EXPO
M
etabolic surgery has an obvious ambiguity. Even
though surgery is most effective treatment for type-2
diabetes mellitus, on the other hand, also has catastrophic
obstacles. Safety concern and postoperative deterioration
of life quality are expected to the major obstacle. Another
expected obstacle is unresponsiveness to surgery or
recurrent hyperglycemia after primary remission. Recently,
we invented new surgical model to cope with possible
obstacles at the same time. Until year 2015, modified
minigastric bypass was primary procedure at our institute.
The mechanism and outcomes were published elsewhere.
Our technique was modified to overcome minor problems
from surgery, even though the results were satisfactory. Until
now, 40 patients were treated with modified technique. The
procedure was conducted under the three principles: Total
duodenal exclusion, securing biliopancreatic limb more
than 200 cm and preservation of pyloric sphincter. Post-
operative quality of life was improved markedly. Meanwhile,
anti-diabetic effect was comparable or even better than
before. We believe that, pyloric sphincter function is critical
to maintain normal GI physiology and possible cause of
recurrent hyperglycemia after primary remission, depends
on the completeness of duodenal exclusion. For example,
the results of the anti-diabetic effect of DJB are quite
inconstant. A group of paper reporting acceptable outcomes
but another group does not. Difference between the groups
is whether using gastrojejunostomy or duodenojejunostomy.
The unique regeneration mechanism of intestinal mucosa
is the clue of enigma. In this presentation the author will
discuss for better anti-diabetic effects with better quality of
life after surgery.
e:
hurusa@hanmail.netFunction preserving metabolic surgery for low BMI type-2 diabetes
Kyung Yul Hur
Soon Chun Hyang University Hospital, South Korea
J Obes Eat Disord, 3:2
DOI: 10.21767/2471-8203-C1-003