neuroimmunology-2017-scientifictracks-abstracts

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Page 30
ISSN:2171-6625
September 18-19, 2017 | Dallas, USA
4th International Conference on
NEUROLOGY AND NEUROIMMUNOLOGY
Notes:
Aage R Møller
The University of Texas at Dallas, USA
Similarities between severe tinnitus and chronic pain
Chronic neuropathic pain and severe tinnitus have many
similarities. Activation of maladaptive neuroplasticity
plays important roles in the creation of the symptoms of both
diseases and they are both plasticity diseases. The symptoms
are generated in the central nervous system. Acute pain that
is caused by tissue injury is often a precursor to chronic
neuropathic pain with stress as a co-factor. While less severe
tinnitus may be generated in the ear, it is believed that
severe tinnitus is caused by changes in the nervous system
that occurs as a result of activation of neuroplasticity. The
changes in the nervous system that produce the symptoms
of these two diseases are altered synaptic efficacy causing a
change in the excitability and functional connections in many
related neural circuits of the brain. These components of the
pathology cannot be detected by the methods currently
available for diagnostic purposes.
Speaker Biography
Aage R Møller is known internationally for his innovative research on sensory systems
and neural plasticity and for developing methods for reducing the risk of neurological
deficits in neurosurgical operations. His work has helped establish UT Dallas as a
leader in tinnitus-related research. His lengthy research career has focused on four
primary areas: The basic function of the ear, sound transmission in the middle ear and
cochlea, the neural code of complex sounds and neural plasticity. He eventually moved
on to research in humans aimed at studying disorders of the year and the nervous
system, such as tinnitus. He began his research career at the famed Karolinska Institut
in Sweden. In 1978, he was invited to join the University of Pittsburgh. There he did
innovative research in the area of neurosurgery and intraoperative neurophysiology;
he developed methods for reducing the risks of serious neurological deficits after
neurosurgical operations. He was one of the founders of a new specialty; intraoperative
neurophysiological monitoring and he did innovative research that lead to better
understanding of several neurological diseases. When he joined UT Dallas in 1997, he
became interested in abnormalities in the nervous system function among individuals
with autism. He developed teaching programs in the biology of pain; sensory systems,
neuroplasticity and he established the first university program in teaching IONM in
a graduate program. During his time at UT Dallas, he was named the university’s
“President’s Teaching Excellence Award,” won Teacher of the Year for the School of
Behavioral and Brain Sciences, and was named distinguished Lecturer in Cognition
and Neuroscience. He earned his PhD in Medical Science at the Karolinska Institut in
Stockholm, Sweden.
e:
Aage R Møller, J Neurol Neurosci, 8:5
DOI: 10.21767/2171-6625-C1-002