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Journal of Alzheimer's & Dementia

About Journal of Alzheimer's & Dementia

The Journal of “Alzheimer's & Dementia” is an international open access journal that publishes peer-reviewed scholary articles on all the areas of neurology which causes Alzheimer's & Dementia. Its goal is to serve international scientific and clinical communities, to strengthen links between science and policy and to stimulate and enhance the quality of debate. This journal will bridge the knowledge gaps across a wide range of bench-to-bedside investigation and encourages original research works to make a significant contribution towards advancing knowledge and supports evidence based clinical practice.

The Journal welcomes submissions from researchers, clinicians and health professionals through out the world who are interested in research, treatment, prevention and care of patients with alzheimer's & dementia.

The Journal encourages research on: Alzheimer's Medicine, Alzheimer Disease, Memory Loss, Alzheimer Treatment, Senile plaque, Beta amyloid, Neurodegenerative disorder, Pathogenesis of Alzheimer’s Disease, Memory Study, Dementia, Biomarkers, Lucid Moments, Alzheimer dementia symptoms, Alzheimer's Incontinence, Dementia Risk, Impaired thinking, Down syndrome, Familial Alzheimer’s disease, Magnetic gait, Mild cognitive impairment (MCI), Parkinson's disease, Schizophrenia and related fields.

The Journal of Alzheimer’s & Dementia publishes the most reliable and complete source of information on the discoveries and current developments in the form of comprehensive reviews, research articles, information on clinical trials; short reports, in-depth perspectives or open-peer commentaries, theoretical papers that attempts to integrate knowledge across discipline, abstracts of papers presented at international meetings and negative results principally clinical trials as short communications, editorials, commentaries, reviews, descriptions, letters & books review, theoretical, state-of-the-science reviews, description.

The journal is using Editorial Manager System for an easy online tracking and managing of the manuscript processing. Each article undergoes a peer-review process under the aegis of an assigned Editor.

Memory Loss

Memory loss, also called Amnesia, happens when a person loses the ability to remember information and events they would normally be able to recall. It could be something that happened seconds or minutes ago, or a memorable event that occurred in the past. It's normal to become a bit forgetful as you get older. Memory loss can be distressing for the person affected, and their family. The memory loss may be for a short time and then resolve (transient). Or, it may not go away, and, depending on the cause, it can get worse over time.

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Senile plaque

A microscopic mass of fragmented and decaying nerve terminals around an amyloid core, numbers of which occur in the brains of people with Alzheimer's disease. Senile plaques are one of the neuropathological hallmarks of Alzheimer's disease (AD) and are found in the extracellular space between neurons in the brain. Fully formed senile plaques are characterized by a central beta-amyloid core and surrounded by elements of degenerating neurons (dystrophic neurites). Senile plaques also occur in the brains of elderly individuals without dementia.

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Alzheimer Treatment

Presently, there is no cure for Alzheimer's. But drug and non-drug treatments may help with both cognitive and behavioral symptoms. Researchers are looking for new treatments to alter the course of the disease and improve the quality of life for people with Alzheimer’s disease. Alternative treatments are Regular exercise, consuming healthy combination of foods and certain nutritional supplements are marketed as "medical foods" specifically to treat Alzheimer's disease. Various herbal mixtures, vitamins and other supplements are widely promoted as preparations that may support cognitive health or prevent or delay Alzheimer's.

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Alzheimer’s Disease

Alzheimer's disease is the most common form of senile dementia. The incidence and prevalence of Alzheimer’s disease rise with increasing age and are higher in women in part because of their increased longevity. The histopathologic features of Alzheimer’s disease were extracellular amyloid plaques and intracellular neurofi brillary tangles. More recently recognized histopathologic features include synaptic degeneration, hippocampal neuronal loss, and aneuploidy.

Related Journal: Neurobiology of Aging Alzheimer's & Dementia Neuroscience Letters Brain Research Journal of the Neurological Sciences Neuroscience Neurobiology of Disease Biochemical and Biophysical Research Communication Free Radical Biology and Medicine Neurochemistry International Journal of Neuroimmunology biochimica et biophysica acta (bba) - molecular basis of disease Experimental Neurology Medical Hypotheses European Neuropsychopharmacology The American Journal of Pathology Biological Psychiatry.

Dementia Risk

Dementia is a general classification of mind sicknesses that cause a long haul and regularly progressive reduction in the capacity to think and recall that is sufficiently awesome to influence a man's day by day functioning. Other basic side effects incorporate passionate issues, issues with dialect, and a diminishing in inspiration. A man's cognizance is generally not affected. A dementia conclusion requires a change from a man's standard mental working and a more prominent decrease than one would anticipate that due will maturing.

Cause 1. Risk genes: it improves the probability of building up a sickness however don't promise it will happen. Analysts have discovered a few qualities that expansion the danger of Alzheimer's. apolipoprotein E-e4, or APOE-e4, is the primary hazard quality recognized and remains the one with most grounded affect. 2. Deterministic genes: Causes a malady, ensuring that any individual who acquires one will build up the turmoil. Researchers have found varieties that straightforwardly cause Alzheimer's infection in the qualities coding three proteins: amyloid antecedent protein

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Down Syndrome

Down disorder is a normally happening chromosomal course of action that has dependably been a piece of the human condition, being all around show crosswise over racial, sex or financial lines, and influencing roughly 1 in 800 live births, despite the fact that there is impressive variety around the world. Down disorder for the most part causes differing degrees of scholarly and physical handicap and related therapeutic issues.

Causes It is caused by an additional duplicate of hereditary material on all or part of the 21st chromosome .Each cell in the body contains qualities that are gathered along chromosomes in the cell's core. There are typically 46 chromosomes in every phone, 23 acquired from the mother and 23 from the father. Whenever a few or the greater part of a man's cells have an additional full, or fractional, duplicate of chromosome 21, the outcome is Down disorder. The most well-known type of down disorder is known as trisomy 21, a condition where people have 47 chromosomes in every cell rather than 46 risomy 21 is caused by a blunder in cell division called nondisjunction, which leaves a sperm or egg cell with an additional duplicate of chromosome 21 preceding or at origination. This variation represents 95 percent of down disorder cases. The rest of the 5% of down disorder cases are because of conditions called mosaicism and translocation. Mosaic Down disorder comes about when a few cells in the body are typical while others have Trisomy. Characteristics are youngsters with down disorder frequently have a discourse delay and require language training to help with expressive dialect. Also, fine engine abilities are deferred and tend to fall behind gross engine aptitudes. Youngsters with down disorder ordinarily linger behind in their formative turning points. The normal time of sitting is 11 months, crawling is 17 months, and strolling is 26 months .Although numerous with the condition encounter formative postponements, individuals with Down disorder go to class and usually end up plainly dynamic, working individuals in the group.

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Impaired Thinking

Gentle subjective debilitation (MCI) causes a slight yet discernible and quantifiable decrease in intellectual capacities, including memory and thinking aptitudes. A man with MCI is at an expanded danger of building up Alzheimer's or another dementia.

Symptoms and causes are in our cerebrum, similar to whatever is left of your body, changes as you become more seasoned. Many individuals see step by step expanding absent mindedness as they age. It might take more time to think about a word or to review a man's name. Be that as it may, steady or expanding worry about your mental execution may propose gentle psychological hindrance (MCI). Intellectual issues may go past what's normal and show conceivable MCI on the off chance that you encounter any or the greater part of the accompanying 1. Depression 2. Irritability and aggression 3. Anxiety 4. Apathy

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Alzheimer Dementia Symptoms

Memory loss that disrupts daily life may be a symptom of Alzheimer's or another dementia. Alzheimer's is a brain disease that causes a slow decline in memory, thinking and reasoning skills. Symptoms of Alzheimer's include Memory loss that disrupts daily life, Confusion with time or place, Trouble understanding visual images and spatial relationships, Misplacing things and losing the ability to retrace steps, Changes in mood and personality. Memory loss and dementia will progress as the stages of Alzheimer's advance. Symptoms can be treated with medication, but there is no cure. Very early stages of Alzheimer’s disease: Mild Alzheimer's disease, Moderate Alzheimer's disease and Severe Alzheimer's disease.

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Beta Amyloid

Beta amyloid is a protein fragment snipped from an amyloid precursor protein (APP). In a healthy brain, these protein fragments are broken down and eliminated. In Alzheimer's disease, the fragments accumulate to form hard, insoluble plaques. Recent research suggests that soluble oligomer forms of the peptide may be causative agents in the development of Alzheimer's disease. It is generally believed that Aβ oligomers are the most toxic. Amyloid beta is commonly thought to be intrinsically unstructured

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Lucid Moments

Lucid Moments are those situations where a person suddenly reacts to the person or the environment by becoming conscious. These moments are temporary and occur rarely in patients with Alzheimer's dementia. Patients with Alzheimer's dementia which occurs at an old age are afflicted with functional, cognitive and behavioural disturbances. The contact with the persons and the environment along with the daily activities are affected.

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Memory Studies

Memory studies are academic field studying the use of memory as a tool for remembering the past. Memory is the past made present and is a contemporary phenomenon, something that, while concerned with the past, happens in the present; and second, that memory is a form of work, working through, labor, or action. The study of memory incorporates research methodologies from neuropsychology, human development and animal testing using a wide range of species.

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Alzheimer’s Disease

Alzheimer’s disease is the most common form of senile dementia. The incidence and prevalence of Alzheimer’s disease rise with increasing age and are higher in women in part because of their increased longevity. The histopathologic features of Alzheimer’s disease were extracellular amyloid plaques and intracellular neurofi brillary tangles. More recently recognized histopathologic features include synaptic degeneration, hippocampal neuronal loss, and aneuploidy.

Related Journal: Neurobiology of Aging Alzheimer's & Dementia Neuroscience Letters Brain Research Journal of the Neurological Sciences Neuroscience Neurobiology of Disease Biochemical and Biophysical Research Communication Free Radical Biology and Medicine Neurochemistry International Journal of Neuroimmunology biochimica et biophysica acta (bba) - molecular basis of disease Experimental Neurology Medical Hypotheses European Neuropsychopharmacology The American Journal of Pathology Biological Psychiatry.

Familial Alzheimer’s Disease

The transformations is available in just a single of the two duplicates of a quality acquired from a man's folks, the individual will unavoidably build up that type of early-onset Alzheimer's (this is called autosomal prevailing legacy). Be that as it may, the aggregate known number of these cases is little (in the vicinity of 100 and 200 around the world), and there is up 'til now no confirmation that any of these changes assume a noteworthy part in the more typical, sporadic or non-familial type generally onset Alzheimer's. Researchers are attempting to uncover the typical capacity of APP and presenilins and to decide how changes of these qualities cause the onset of familial Alzheimer's ailment.

Causes Hereditary transformation-a perpetual change in a quality that can be passed on to youngsters uncommon, early-onset familial type of Alzheimer's infection is related with transformations in qualities on chromosomes 21, 14, and 1. Hereditary hazard consider—An adjustment in a quality that builds a man's danger of building up an ailment

Signs of alzheimers disease are Memory loss that disrupts daily life Challenges in planning or solving problems Confusion with time or place New problems with words in speaking or writing Decreased or poor judgment

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Mild cognitive impairment (MCI)

Mild cognitive impairment (MCI) was proposed as a nosological entity referring to elderly people with mild cognitive deficit but no dementia. MCI is a heterogeneous clinical entity with multiple sources of heterogeneity. The concept of MCI was reviewed and a diagnostic procedure with three different stages was proposed by the European Consortium on Alzheimer’s Disease Working Group on MCI. Firstly, MCI should correspond to cognitive complaints coming from the patients or their families; the reporting of a relative decline in cognitive functioning during the past year by a patient or informant; cognitive disorders as evidenced by clinical evaluation; absence of major repercussions on daily life; and absence of dementia. These criteria, similar to those defined during an international workshop in Stockholm, make it possible to identify an MCI syndrome, which is the first stage of the diagnostic procedure. Secondly, subtypes of MCI had to be recognised. Finally, the aetiopathogenic subtype could be identified. Identifying patients at a high risk for progression to dementia and establishing more specific and adapted therapeutic strategies at an early stage, together with more structured overall management, is made possible by the diagnostic procedure proposed.

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Alzheimer's Incontinence

Alzheimer's disease is the most widely recognized type of dementia, which is caused by a physical malady of the mind. It is a dynamic sickness, which causes loss of memory, judgment and dialect. As a man's condition decays, they regularly pull back from family and society. Progressively, substantial capacities are lost, at last prompting death. Although the speed of movement can fluctuate, the normal future after conclusion is three to nine years.

The symptoms of this disease are

• Memory misfortune individual may overlook where they have left things, for example, their keys

• May battle with finding the correct words amid discussions, rehash themselves or overlook individuals' names

• Lose all sense of direction in a natural place

Causes: Incontinence can be caused by many factors, including an inability to recognize the need to use the restroom, forgetting where the bathroom is located, medications, stress, certain physical conditions, clothing that is difficult to remove and constipation.

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Magnetic Gait

The cardinal sign of NPH (Normal pressure hydrocephalus) is a hypokinetic gait disorder in which the older adult's feet look as though they are glued to the floor. The gait also has been described as magnetic. People with NPH also may have mild dementia and bladder and bowel incontinence.

• Feet seem attached to floor as if by a magnet. In magnetic gait, each step is initiated in a "wresting" motion carrying feet upward and forward. Magnetic gait can be visualized in terms of a powerful magnet being forcefully pulled from a steel plate. Symptoms- A particular walk where feet appear to be held tightly to the floor

• Stomping walk.

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Journal Highlights
  • Memory Loss
  • Senile plaque
  • Alzheimer Treatment
  • Alzheimer’s Disease
  • Dementia Risk
  • Down Syndrome
  • Impaired Thinking
  • Alzheimer Dementia Symptoms
  • Beta Amyloid
  • Lucid Moments
  • Memory Studies
  • Alzheimer’s Disease
  • Familial Alzheimer’s Disease
  • Mild cognitive impairment (MCI)
  • Alzheimer's Incontinence
  • Magnetic Gait
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